Evaluation
Because elderly persons frequently fail to report falls, they should be asked about them as a routine part of screenings. When a patient does report a fall, the first step is to assess and, if necessary, treat any acute injury. Then the fall should be evaluated like any symptom, with a history and physical examination (see TABLE 7-3). The routine use of laboratory tests, electrocardiograms, and Holter monitoring is not recommended unless a specific clinical indication exists. The evaluation of a fall may also include an assessment of environmental hazards. The evaluation of falls associated with syncope differs from that of falls without syncope (see also Ch. 6).
History: The initial inquiry concerning the circumstances of a fall should be made using open-ended questions. Then more specific questions should be asked about when and where the fall occurred and what the patient was doing. Did the patient experience any symptoms such as palpitations, shortness of breath, chest pain, vertigo, or lightheadedness at the time of the fall? Did the patient lose consciousness? Were any obvious environmental hazards involved? Does the patient have a history of falls? The history should also explore present and past medical problems and medication use. Any witnesses to the fall should be questioned as well.
Physical examination: The physical examination should be comprehensive enough to exclude obvious intrinsic causes of falling and to help distinguish syncope from other causes, such as tripping. The pattern of injury is useful, but not conclusive, evidence in determining the cause.
Emphasis should be placed on examining the cardiovascular, musculoskeletal, and neurologic systems. Blood pressure should be measured with the patient both supine and standing to rule out orthostatic hypotension. Examining the cardiovascular system helps exclude arrhythmia, valvular heart disease, and heart failure. The extremities should be evaluated for arthritis and podiatric problems that could impair gait. Neurologic findings may suggest cerebrovascular disease, tumor, Parkinson’s disease, myelopathy, peripheral neuropathy, or proximal myopathy. Mental status testing may suggest dementia, delirium, or depression. Vision and hearing should be checked as well. Because falling may be an atypical presentation of an acute illness, occult problems such as infection, myocardial infarction, dehydration, and anemia also should be reasonably evaluated.
A set of simple clinical tests, in which patients perform position changes and gait maneuvers during daily activities, can identify those at greatest risk of falling. This type of evaluation is more likely than a formal neurologic examination to detect fall-related risk factors. The patient is observed getting into and out of a chair, turning around, bending down and picking up objects from the floor, and reaching up to get something from a shelf. Aside from revealing abnormalities that might not have been apparent on a routine inspection of gait (eg, proximal muscle weakness that may be apparent only when the patient rises from a chair), this testing also helps establish functional abilities.
Gait should be observed with the patient rising from a chair, walking at least 20 ft, turning, returning, and sitting down. The clinician can walk alongside the patient for safety. In the circumduction gait, the lower extremity assumes triple extension at the hip, knee, and ankle,and the person swings the leg in an outward arc to ensure ground clearance. This type of gait occurs in patients with hemiplegia. Bilateral upper motor neuron lesions may cause a scissoring gait, which is essentially a bilateral circumduction gait.
The festinating gait is a symmetric shuffling of the feet with poor ground clearance, typically seen in Parkinson’s disease. Frequently, festination is seen only when the person starts to walk and when the person reaches an obstacle or attempts to turn. The parkinsonian patient also tends to assume a forward-flexed posture and have little or no arm movement while walking. Severe postural instability in the parkinsonian patient leads to an inability to maintain balance when pushed from the back or front, known as propulsion and retropulsion, respectively.
The cerebellar gait is a broad-based gait with irregular steps. The patient veers to either side, forward, or backward. In severe cases, the patient is not able to stand unsupported, even with open eyes. This form of ataxia is common in chronic alcoholics and in persons with paleocerebellar atrophies, progressive supranuclear palsy, multiple sclerosis, and cerebellar tumors. When such ataxia has an acute onset in an older person, the cause is almost always vascular.
Frontal lobe apraxia causes a broad-based gait that in many respects resembles the parkinsonian gait. The person assumes a forward-flexed posture, and the steps are short, slow, and shuffling; at times, the feet appear to be glued to the floor. However, a bedside examination reveals normal power in the legs, and the patient may be able to perform complex movements with the legs, such as drawing a figure 8 on the floor. This gait disorder frequently precedes the dementia and incontinence that complete the triad of normal-pressure hydrocephalus. Because this neurologic condition is potentially treatable with a shunt procedure, it should be considered when a patient presents with an apraxic gait.
Sensory ataxia is a broad-based, foot-stamping gait. The person constantly looks at his feet as he walks to compensate for the lack of proprioception from visual input. When asked to stand with his feet together and eyes open, the patient can maintain the stance, but when asked to close the eyes, the patient loses balance—a positive Romberg test. In the elderly, this condition is caused by disorders affecting the posterior columns, such as vitamin Bn deficiency (posterolateral sclerosis), cervical spondylosis, and paleocerebellar degeneration.
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